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Злокачественные опухоли

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РОЛЬ СТРЕСС-ИНДУЦИРОВАННЫХ МОЛЕКУЛ MICA/B В ПРОТИВООПУХОЛЕВОМ ИММУННОМ ОТВЕТЕ

https://doi.org/10.18027/2224-5057-2012-2-102-104

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Е. В. Абакушина
ФГБУ Медицинский радиологический научный центр Минздрава России, Москва
Россия


Список литературы

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4. Bauer S, Groh V, Wu J. et al. Activation of NK cells and T cells by NKG2D, a receptor for stress-inducible MICA. Science 1999, 30, 285, 5428, 727-729.

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9. Salih HR, Antropius H, Gieseke F, et al. Functional expression and release of ligands for the activating immunoreceptor NKG2D in leukemia. Blood 2003; 102: 1389–1396.

10. Holdenrieder S., Stieber P., Peterfi A., et al. Soluble MICA in malignant diseases. Int J Cancer 2006, 1, 118(3): 684-7.

11. Zwirner N, Fuertes M, Girart M-V, et al. Cytokinedriven regulation of NK cell function in tumor immunity: role of the MICA-NKG2D system. Cytokine and growth factor reviews 2007, 18:159-170.

12. Kenneth F. May. Isolation of human anti-MICA antibody from cancer patient responding to immunotherapies. June 2012, ASCO Annual meeting.


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Абакушина Е.В. РОЛЬ СТРЕСС-ИНДУЦИРОВАННЫХ МОЛЕКУЛ MICA/B В ПРОТИВООПУХОЛЕВОМ ИММУННОМ ОТВЕТЕ. Злокачественные опухоли. 2012;2(2):102-104. https://doi.org/10.18027/2224-5057-2012-2-102-104

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ISSN 2224-5057 (Print)
ISSN 2587-6813 (Online)